Simalip, you're correct in saying there is an increased risk of developing cancer if you have Barrett's Oesophagus and I am aware the lifetime risk shows increased chance over 20 years.
Oesophageal Adenocarcinoma is currently the 8th most prevalent cancer and the number 5 cancer killer in the UK which has the highest per capita rate of this in the world.
It's identifiable precursor lesion is Barrett's Oesophagus which is what the charity I co-founded and chair in UK, Barrett's Wessex, focusses on. "Our principal aim is to reduce the number of deaths to oesophageal cancer through raising awareness of its predominant pre-cancerous lesion, Barrett's Oesophagus."
In US some years ago, based on cadaver studies, the Ryan Hill foundation discovered at death (from whatever cause), 1 person in 70 of the population had Barrett's Oesophagus though only 10% knew it.
Transferring that figue to UK would mean we have 1 million with the disease and 8000 a year are dying here from OAC. However, I agree figures may actually be grossly underestimated. US has 5 times the population of UK but only twice the number of annual deaths from OAC so it may be in UK we could even have 2.5 million with Barrett's Oesophagus.
However, we must not be alarmist. Those diagnosed with Barrett's can be considered the lucky ones as regular surveillance scoping every few years will identify any pre-cancerous dysplastic changes early enough to be ablated. It's those who are unaware they have it who are likely not to know until the condition has actually become cancerous when it may be too late with a prognosis measured in weeks rather than years.
The most recent risk figures we commissioned from the statistics branch of cancer Research UK last year which took into account numerous studies in Europe and America, shows an annual progression risk of 0.7% from Non-Dysplastic Barrett's, 1% from Low Grade Dysplasia and 6% from High Grade Dysplsia.
Barrett's Oesophagus is a cellular change from squamous cells to columnar cells as a protection to the body to prevent it digesting itself.
Both Acid and bile are both needed. Bile acts as the surfactant required for the acid to attack animal tissue and fats.
A study from University of Rochester NY published August 2013, found "bile at low pH, but not bile or low pH alone" is required.
To reduce risk of development of Barrett's, we need to control at least one of the elements of acid, bile or reflux. The easiest is to reduce acid using PPIs.
The Keck video you link to is actually a promotion of the LINX device which will not stop the possibility of progression to cancer if it is to occur - in the same way as I know my fundoplication will not stop possible progression of mine.
To minimise the risks of progression, Barrett's cells may be ablated. However, since this will not remove the underlying cause for the Barrett's to have developed initially, acid suppressant medication or reflux reduction surgery is required subsequent to completing a course of ablation therapy and continuation of surveillance.